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Polymorphism and protein expression of MUTYH gene for risk of rheumatoid arthritis | BMC Musculoskeletal Disorders | Full Text
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Diagram of the MUTYH protein in scale. Known functional domains and... | Download Scientific Diagram
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Association of Functional Variants and Protein-to-Protein Physical Interactions of Human MutY homolog linked with Familial Adenomatous Polyposis and colorectal cancer Syndrome | bioRxiv
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23andMe says you carry a variant in the MUTYH gene. Now what? - Gene Cuisine: satisfying your appetite for genetic news and information
![Loss of MUTYH function in human cells leads to accumulation of oxidative damage and genetic instability | Oncogene Loss of MUTYH function in human cells leads to accumulation of oxidative damage and genetic instability | Oncogene](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fonc.2012.479/MediaObjects/41388_2013_Article_BFonc2012479_Fig1_HTML.jpg)
Loss of MUTYH function in human cells leads to accumulation of oxidative damage and genetic instability | Oncogene
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IJMS | Free Full-Text | Oxidative Damage in Sporadic Colorectal Cancer: Molecular Mapping of Base Excision Repair Glycosylases in Colorectal Cancer Patients
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Distinct functional consequences of MUTYH variants associated with colorectal cancer: Damaged DNA affinity, glycosylase activity and interaction with PCNA and Hus1. - Abstract - Europe PMC
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DNA glycosylase encoded by MUTYH functions as a molecular switch for programmed cell death under oxidative stress to suppress tumorigenesis - Oka - 2011 - Cancer Science - Wiley Online Library
The Polymorphic AluYb8 Insertion in the MUTYH Gene is Associated with Reduced Type 1 Protein Expression and Reduced Mitochondrial DNA Content | PLOS ONE
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Distinct functional consequences of MUTYH variants associated with colorectal cancer: Damaged DNA affinity, glycosylase activity and interaction with PCNA and Hus1 - ScienceDirect
![DNA glycosylase encoded by MUTYH functions as a molecular switch for programmed cell death under oxidative stress to suppress tumorigenesis - Oka - 2011 - Cancer Science - Wiley Online Library DNA glycosylase encoded by MUTYH functions as a molecular switch for programmed cell death under oxidative stress to suppress tumorigenesis - Oka - 2011 - Cancer Science - Wiley Online Library](https://onlinelibrary.wiley.com/cms/asset/7291bc2f-df15-42b9-b7f7-cb42f6d05fff/cas_1869_f4.gif)